Final answer:
The presence of mitogens typically results in the activation of Cdc25, which facilitates cell cycle progression. Additionally, HPV's E6 protein binds to p53, marking it for degradation, and compromising its tumor-suppressing functions. Option e is the answer.
Step-by-step explanation:
The presence of mitogens normally results in the activation of Cdc25. Mitogens are substances that stimulate cell division. In terms of cell cycle regulation, Cdc25 is a phosphatase that activates Cyclin-dependent kinases (Cdks) by dephosphorylating them, which then promotes the cell cycle progression. Furthermore, the Human papillomavirus (HPV) encodes E6, a protein that binds to and leads to the degradation of p53, a tumor suppressor gene product. By doing so, E6 contributes to the potential development of cervical cancer as p53 normally acts to suppress the formation of tumors by stopping the cell cycle, aiding DNA repair, or initiating cell death when DNA repair is not possible.