Question

Sometimes after an intense exercise routine, an individual will have sore muscles 1 to 2 days following the exercise; this is called delayed onset muscle soreness. Although not completely understood, it is believed that one cause of this is damage that occurs to the Z disks and contractile filaments in the muscles. When the body attempts to repair this damage, an inflammatory response occurs that causes soreness a couple of days after the exercise has taken place. Which of these situations would likely lead to less delayed onset muscle soreness in an individual?

a. a mutation in the Z-disk protein that caused the attachment of the Z disk to the titin protein to be stronger
b. a mutation that would reduce the amount of lactic acid buildup in muscle tissue
c. a mutation in the Z-disk protein that caused the attachment of the Z disk to the titin protein to be weaker
d. a mutation that weakens the calcium binding ability of troponin

Answer 1

The correct answer is option A. a mutation in the Z-disk protein that caused the attachment of the Z disk to the titin protein to be stronger

Step-by-step explanation:

As delayed onset muscle soreness is thought to be led by the inflammatory response of the body in order to repair damage to the Z disks and contractile filaments in muscles, due to the intense exercise.

Mutation in the Z-disk protein would strengthen the connection between Z disks and contractile filaments as it is logical that this mutation could withstand more pressure or intense exercise before damage occurs to these tissues.