Final answer:
The statement regarding the subfornical organ stimulating ADH release is false. ADH release is mainly stimulated by high plasma osmolarity or low blood volume, and it is controlled by the hypothalamus and released from the posterior pituitary gland to act on the kidneys.
Step-by-step explanation:
The statement that impulses from the subfornical organ stimulate the release of ADH is false. The primary stimuli for the release of ADH, or antidiuretic hormone, are related to the osmotic pressure of the plasma (oncotic pressure) and blood volume.
When plasma is hypertonic, which means it has a high concentration of solutes, or when blood volume is low, the secretion of ADH increases. The hypothalamus is the region of the brain that produces ADH and controls its release into the bloodstream, primarily in response to these changes in osmolarity and blood volume.
ADH is then transported to and released from the posterior pituitary gland, influencing the kidneys to reabsorb more water, thus reducing urine volume and increasing blood pressure. Dehydration or physiological stress can boost osmolarity and trigger more secretion of this hormone.
On the other hand, certain substances, like alcohol, can inhibit the release of ADH, leading to greater urine production and potential dehydration.