Final answer:
The release of aldosterone causes the kidneys to reabsorb sodium, leading to water retention and increased blood volume, not directly increasing blood colloid osmotic pressure. The statement in question is false.
Step-by-step explanation:
The release of aldosterone can cause decreased urine output by prompting the kidneys to reabsorb sodium, leading to water retention and an increase in blood volume.
However, contrary to the suggestion in the question, aldosterone does not directly increase the blood colloid osmotic pressure, which is primarily determined by the concentration of plasma proteins. Instead, aldosterone's action on reabsorbing sodium and subsequent water retention could lead to an increase in overall blood pressure.
When blood pressure decreases, the renin-angiotensin-aldosterone system (RAAS) is activated. Cells in the juxtaglomerular apparatus detect the low blood pressure and release renin, which facilitates the production of angiotensin II.
Angiotensin II then stimulates aldosterone release from the adrenal cortex, which ultimately results in increased sodium reabsorption and water retention, thereby increasing blood pressure.
Therefore, the statement 'The release of aldosterone can cause an increase in the blood colloid osmotic pressure' is false.