Final answer:
The prompt increase in the concentration of certain inhalational anesthetics can cause a sympathomimetic response, resulting in an elevation of heart rate, bronchodilation, constriction of blood vessels, and enhanced gluconeogenesis.
Step-by-step explanation:
Understanding Sympathetic Response to Inhalational Anesthetics
Upon rapid increase in inspired concentration of certain inhalational anesthetics, there may be an elicitation of a sympathetic response. Anesthetics can stimulate the sympathetic nervous system, which, when activated, releases adrenaline (epinephrine). This leads to a series of effects: increasing heart rate and contractility, causing blood vessels to constrict, acting as a bronchodilator by dilating the bronchi of the lungs thereby increasing air volume, and stimulating gluconeogenesis. These physiological changes represent the body's fight-or-flight response, optimized to prepare an individual for action.
Fast induction of anesthesia with these agents may provoke such sympathomimetic effects, mimicking the natural release of adrenaline, potentially leading to increased blood pressure and hyperpnea, which is an increased rate and depth of breathing due to heightened oxygen demand, without significantly altering blood gases.