Final answer:
High-density lipoproteins (HDL) are known to inhibit platelet aggregation at plaque sites by removing cholesterol from the arteries and transporting it back to the liver.
Step-by-step explanation:
High-density lipoproteins (HDL) inhibit platelet aggregation at plaque sites. Unlike low-density lipoproteins (LDL), which contribute to the buildup of plaques in arteries, HDL works to remove cholesterol from tissues, including the arterial walls, and transport it back to the liver for excretion. This function has earned HDL the nickname of "good cholesterol," and it helps prevent the progression of atherosclerosis. Chylomicrons, on the other hand, primarily transport dietary lipids from the intestines to other locations in the body and are not directly involved in platelet aggregation inhibition. Therefore, among the options provided, only high-density lipoprotein has a role in inhibiting platelet aggregation at the site of the plaque.