Final answer:
A decrease in urine output and improvement in excessive thirst indicate effective treatment with aqueous vasopressin for diabetes insipidus. Vasopressin facilitates water reabsorption in the kidneys, reducing symptoms. Treatment efficacy is assessed by urine volume and patient reports.
Step-by-step explanation:
The effective treatment for a client with diabetes insipidus (DI) prescribed aqueous vasopressin would be indicated by a decrease in urine output and an improvement in symptoms such as excessive thirst (polydipsia) and frequent urination (polyuria). Diabetes insipidus involves a deficiency in antidiuretic hormone (ADH), also known as vasopressin, which results in an inability of the kidneys to conserve water, leading to large volumes of dilute urine and increased thirst. As ADH play a crucial role in the body's regulation of water, proper function regained through treatment with vasopressin should counter the excessive loss of water, normalize urine volume, and mitigate dehydration risks.
Vasopressin, or ADH, achieves its effect by stimulating the movement of aquaporin proteins into the cell membrane of kidney ducts, effectively allowing the body to reabsorb more water and reduce urine output. When aqueous vasopressin is administered, it mimics the action of natural ADH, thereby mitigating the symptoms of DI. The effectiveness of treatment can be assessed by monitoring urine volume and concentration as well as patient reports of decreased thirst.