Final answer:
The claim that insulin reduces glucose uptake and storage in fat cells is false; insulin, in fact, increases the uptake and storage of glucose in cells, including liver and muscle cells, and promotes the conversion of glucose into glycogen and fatty acids.
Step-by-step explanation:
The statement in the question is false. Insulin increases glucose uptake and storage among fat cells. Insulin binds to its receptor and facilitates a cascade of protein activations which leads to several cellular actions. Notably, this promotes the translocation of Glut-4 transporters to the plasma membrane, allowing glucose to enter cells through facilitated diffusion. Once inside cells, glucose is converted to glucose-6-phosphate to maintain a concentration gradient that favors glucose entry into cells.
Additionally, insulin stimulates the liver to produce glycogen from glucose for storage, inhibits the breakdown of stored glycogen into glucose, and inhibits the production of glucose from non-carbohydrate sources. Insulin also promotes the synthesis of fatty acids and protein. After a high-glucose meal, insulin helps to decrease blood glucose levels by enabling both the transport and storage of glucose, not increase blood glucose levels as stated in some incorrect options. Consequently, the function of insulin is crucial for maintaining proper blood glucose levels and energy storage within the body.