Final answer:
In the context of injury-induced cataract formation, monocytes would actively utilize TGF transporters and receptors, along with cytokines, as part of the healing and immune response. Tight junctions, however, are structures associated with epithelial cells and are the least likely to be found in monocytes.
Step-by-step explanation:
The question addresses the changes in monocytes following injury-induced cataract formation. Cataracts involve the clouding of the eye's lens, which can be caused by injury among other factors. Monocytes are white blood cells that play a role in inflammation and tissue repair processes.
Regarding the options provided:
- TGF (Transforming Growth Factor) transporters and TGF receptors would be expected in monocytes, as TGF signalling is often involved in healing and immunoregulatory processes.
- Cytokines are signalling molecules used in immune responses. They are expected in and around monocytes during inflammation and tissue damage.
- Tight junctions are cell-to-cell adhesion structures typically found in epithelial cells, which are quite different from monocytes. Their main purpose is to prevent the passage of materials between the cells they connect. Therefore, tight junctions are not characteristic of monocytes, especially when compared to the other options given that are directly involved in monocyte function.
So, tight junctions are the least likely to be found in nearby monocytes after injury-induced cataract formation begins.