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Concerning the role of hypothalamus and the posterior pituitary gland in osmoregulation and especially when water potential of the blood is low, When the osmoreceptors in the hypothalamus detect the low water potential of the blood, they lose water by osmosis and shrink, does this shrinkage stimulates the syntheisis of the ADH in the neurosecretory cells in hypothalamus first and then ADH is sent along the axons where they terminate in the posterior pituitary gland secondly and released into the blood stream thirdly ?

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Final answer:

When the blood water potential is low, the osmoreceptors in the hypothalamus shrink and stimulate the synthesis of ADH. ADH is then stored in the posterior pituitary gland and later released into the bloodstream to regulate water reabsorption in the kidneys.

Step-by-step explanation:

When the blood water potential is low, the osmoreceptors in the hypothalamus shrink due to water loss by osmosis. This shrinkage stimulates the synthesis of antidiuretic hormone (ADH) in the neurosecretory cells of the hypothalamus. ADH is then sent along the axons and stored in the posterior pituitary gland. Finally, when needed, ADH is released into the bloodstream to regulate water reabsorption in the kidneys.

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