Final answer:
The CP-symmetry complex phase is derived from fitting data related to the CKM matrix, using models that include CP violation. In cellular models, the nematic order parameter P₁ is predicted by the Maier-Saupe theory and is dependent on cell shape, substrate rigidity, and elastic forces within the cytoskeleton and matrix.
Step-by-step explanation:
The process of extracting the CP-symmetry complex phase from data involves several stages, including signal selection, data filtering, and fitting to a theoretical model. To theoretically determine the CP-symmetry complex phase, one must use the parameters related to the Cabibbo-Kobayashi-Maskawa (CKM) matrix. These parameters are obtained by fitting experimental data to phenomenological models of particle interactions that account for CP violation. In the context provided, matrix refers to the forces induced by the deformation of the matrix due to dipoles, affecting the organization of the cytoskeleton (CSK) but not the magnitude of dipoles. Long-range elastic signals, predicted by the real-space Eshelby formalism, lead to the nematic order of internal force dipoles. The nematic order parameter, P₁, is obtained through a self-consistent prediction based on the cell and matrix elastic constants, cell shape, and effective temperature. The Maier-Saupe theory is used to predict the nematic order parameter, P₁, highlighting the importance of long-range elastic interactions. This is in contrast with molecular systems where interactions are typically short-range. The determined nematic order parameter reflects the behavior cells on substrates of varying rigidity and shapes.