Final answer:
Cocaine is the chemical that blocks neuronal uptake of noradrenaline, acting as a reuptake inhibitor. It leads to increased noradrenaline levels in the synaptic cleft, prolonging its action.
Step-by-step explanation:
The chemical that blocks neuronal uptake of noradrenaline is cocaine. Cocaine acts as a reuptake inhibitor for several neurotransmitters, including noradrenaline, by binding to the transporter proteins and preventing the reabsorption of these neurotransmitters back into the presynaptic neuron. This leads to an increase in the concentration of neurotransmitters like noradrenaline in the synaptic cleft, intensifying their effects on the postsynaptic neuron.
For other related neurotransmitter questions:
- Acetylcholine is the neurotransmitter that results in the constriction of the pupil.
- A drug that affects both divisions of the autonomic system would likely interact with nicotinic or muscarinic neurotransmitter receptors.
- An agonist is a drug that activates a receptor, whereas an antagonist blocks the receptor.
Regarding paracrine chemicals causing vasodilation, nitric oxide is a well-known vasodilator.
At the neuromuscular junction, the neurotransmitter released is acetylcholine, not norepinephrine or serotonin.
The drug used to counteract the effects of anaphylactic shock is epinephrine, which is also an example of an agonist, as it stimulates the same receptors as the naturally occurring neurotransmitter.