Final answer:
Devin was sent home as she was experiencing Braxton Hicks contractions, not true labor. Janine was induced with pitocin due to her post-term pregnancy. Premature infants require supplemental oxygen due to immature lungs, and abrupt cessation of breastfeeding can lead to breast engorgement.
Step-by-step explanation:
Understanding Labor and Prenatal Development
When Devin presented at 35 weeks pregnant with signs of labor, the absence of cervical dilation and an intact mucus plug indicated that she was not in true labor. True labor is characterized by progressive cervical dilation and effacement, often accompanied by the rupture of the mucus plug or 'bloody show'. The contractions she felt were likely Braxton Hicks contractions, which are not a true indication of labor. Therefore, she was advised to return home and monitor her symptoms.
In contrast, Janine, who was 41 weeks pregnant with mild but irregular contractions and minimal cervical dilation, was at risk for post-term pregnancy complications. The use of pitocin was to induce labor, as the pregnancy had surpassed the typical gestational period of 40 weeks, and there was a need to stimulate stronger contractions and labor progression to reduce potential risks for both mother and child.
Premature infants may require supplemental oxygen due to underdeveloped lungs. The newborn's first breath triggers a significant change in the circulatory pattern, shifting from fetal circulation, which bypasses the lungs, to normal circulation where blood flows through the lungs for oxygenation.
Breastfeeding involves the transit of milk from the lactocytes through the milk ducts to the nipple pores for the infant to feed. Sudden cessation of breastfeeding can lead to breast engorgement and leakage due to the buildup of milk and pressure in the milk ducts. For studies involving genetics, such as Mendel's experiments, a large sample size is essential for conclusive and reliable results, minimizing the impact of random chance.
An X-linked recessive disorder can affect a female child if she inherits the defective X chromosome from both parents—her mother being a carrier and her father having the disorder or being a carrier as well.