Final answer:
The movement of dynein arms attached to the A tubules of the outer doublets walking along the B tubules of the adjacent doublet allows a flagellum to make a whip-like motion.
Step-by-step explanation:
The property that allows a flagellum to make a whip-like motion is the movement of the dynein arms attached to the A tubules of the outer doublets walking along the B tubules of the adjacent doublet. When the doublets on one side of an axoneme take a walk while the ones on the other side hold still, the microtubules slide past one another, causing the flagellum to bend.
The whip-like motion of a flagellum is caused by the activity of dynein motor proteins walking along microtubules, using energy from ATP hydrolysis, resulting in the bending of the axoneme.
The property that allows a flagellum to make a whip-like motion is the movement of dynein arms attached to the A tubules of the outer doublets walking along the B tubules of the adjacent doublet. This action is empowered by the hydrolysis of ATP. When dynein arms on one side of the axoneme become activated while those on the other side remain stationary, the axoneme bends due to the sliding of microtubules past one another. The movement is regulated by the flexible nexin and radial spoke, which constrain the microtubule sliding, thus resulting in the bending motion characteristic of eukaryotic flagella.
In eukaryotic cells, dynein motor proteins use the energy from ATP hydrolysis to move along the microtubules, generating the force required for the bending motion. This mechanism is fundamentally different from the rotation observed in bacterial flagella, which involves a rigid structure and a rotational motor instead of the bending wave-like motion seen in eukaryotic flagella.