Final answer:
The action of telomerase that adds nucleotide sequences to the lagging strand at chromosome ends does not create 3'-protruding ends for strand invasion; such ends are generated through a different mechanism involving the homologous recombination repair process.
Step-by-step explanation:
The series of events that do not generate 3'-protruding ends for strand invasion are related to the removal of primers and the replacement with DNA on the lagging strand. The process that does lead to 3'-protruding ends involves additional proteins that facilitate the strand invasion, which is part of the homologous recombination repair mechanism. During this process, enzymes like helicase and RNA primase work together to allow the lagging strand to be replicated in short, discontinuous fragments, which are later joined by DNA ligase. However, the action of telomerase at the ends of linear chromosomes, adding nucleotide sequences to the lagging strand to prevent loss of genetic information, does not constitute the generation of 3'-protruding ends for strand invasion. Instead, it prevents the shortening of chromosomal ends during DNA replication.