Final answer:
The child with both copies of chromosome 15 from the father and none from the mother would have Prader-Willi syndrome, as this condition is caused by the absence of certain genes that should be inherited from the mother's chromosome 15. Angelman syndrome is not the correct answer because it typically results from deletions or issues with the maternal copy. In this case, Beckwith-Wiedemann syndrome and normal phenotype are not applicable.
Step-by-step explanation:
If a sperm from the father with two copies of chromosome 15 fertilizes an egg from the mother that has no copies of chromosome 15, the conceptus would indeed have two copies of chromosome 15, which is the normal disomic complement. However, the resulting phenotype of the child depends on whether the specific genes on chromosome 15 are subject to imprinting, a process where only one allele (either the maternal or the paternal) is expressed while the other is silenced.
Angelman syndrome and Prader-Willi syndrome are two conditions associated with imprinting and the deletion or uniparental disomy (UPD) of chromosome 15. Angelman syndrome is typically caused by a deletion in the maternal chromosome 15 or paternal UPD, meaning both copies of the affected region come from the father. On the other hand, Prader-Willi syndrome is caused by a deletion in the paternal chromosome 15 or maternal UPD.
Considering the scenario where the conceptus has received both copies of chromosome 15 from the father and none from the mother, it would lead to the development of Prader-Willi syndrome (C), given that the required genes from the mother's side are absent and the paternal genes are not imprinted (silenced).
Beckwith-Wiedemann syndrome, though associated with genetic abnormalities, is not typically related to chromosome 15, so it does not fit the described situation. The option (A) normal is also incorrect in this context, as the uniparental disomy leads to an abnormal phenotype due to imprinting effects.