Final Answer:
Magnesium toxicity, a concern in preterm labor, manifests early as reduced deep tendon reflexes due to its CNS depressant effect. Adequate urinary output, normal respiration, and the absence of tachycardia make hyporeflexia the key indicator. So, the correct option is B) Hyporeflexia.
Step-by-step explanation:
Magnesium sulfate is commonly administered to manage preterm labor, as it helps prevent seizures. However, excessive magnesium levels can lead to toxicity, necessitating vigilant monitoring of signs and symptoms. Among the options provided, hyporeflexia (Option B) is indicative of magnesium toxicity. Magnesium acts as a central nervous system depressant, and one of the early manifestations of its excessive presence is the reduced or absent deep tendon reflexes, known as hyporeflexia. This occurs due to the inhibitory effect of magnesium on neuromuscular transmission.
Contrarily, options A, C, and D are inconsistent with expected signs of magnesium toxicity. Adequate urinary output of 60 mL/hr (Option A) is a positive indication, ruling out impaired renal function associated with magnesium toxicity. A respiratory rate of 14/min (Option C) falls within the normal range, and tachycardia (Option D) is not a typical feature of magnesium toxicity.
In summary, recognizing hyporeflexia is crucial in identifying magnesium toxicity during preterm labor management. This sign reflects the drug's impact on the nervous system and prompts timely intervention to prevent further complications.