Final answer:
Among the options listed, insulin is the hormone that does NOT increase cardiac contractility, whereas epinephrine, thyroid hormones, glucagon, and angiotensin II have an increasing effect on heart muscle contraction strength.
Step-by-step explanation:
The hormone that does NOT increase cardiac contractility among the given options is D) insulin. Insulin primarily regulates glucose metabolism and has no direct effects on increasing the strength of heart muscle contraction. On the other hand, epinephrine and thyroid hormones do increase cardiac contractility, as they are both strong positive inotropic agents. Glucagon, released by the pancreas, also has a positive inotropic effect, much like epinephrine and norepinephrine. Lastly, angiotensin II, which is not listed here as a direct inotropic agent, primarily works as a vasoconstrictor and can indirectly increase cardiac contractility by increasing afterload.