Final answer:
J. S. is likely developing acute renal failure due to blood loss and kidney hypoperfusion. Untreated, it can progress to end-stage renal disease. Immediate fluid and blood product replacement, along with monitoring of renal function tests, is necessary for treatment.
Step-by-step explanation:
J. S. is likely developing a form of acute renal failure, also known as acute kidney injury (AKI), due to the significant loss of blood and the subsequent hypoperfusion of the kidneys. The low urine output (oliguria), high urine osmolality, and low urine sodium are indicative that the kidneys are trying to conserve water and sodium, reflecting a state of shock and renal conservation in response to acute injury. Without adequate treatment, J. S. may progress to chronic kidney disease or end-stage renal disease (ESRD), where the kidneys can no longer function effectively on their own. To prevent this, J. S. needs immediate fluid and blood product replacement to restore intravascular volume and correct the hypoperfusion. Additionally, other renal function tests should be collected, such as serum creatinine, blood urea nitrogen (BUN), and electrolytes.