Final answer:
In cells that divide infrequently, like neurons or liver cells, cyclin G1 would accumulate at a low rate or not at all, due to these cells often being in a quiescent Go phase where they are not preparing to divide and there's no progression of the typical cell cycle mechanisms.
Step-by-step explanation:
In cells that divide infrequently, such as neurons or liver cells, the accumulation of cyclin G1 would be expected to be quite slow. These cells often reside in the Go phase, a quiescent phase where they are not actively preparing to divide. The Go phase can be permanent for such cells, which means that cyclin G1, a protein that helps manage the cell cycle, might accumulate in very low levels, or potentially not at all if the cells do not re-enter the cell cycle. In highly specialized cells like cardiac muscle cells or cortical neurons, which very rarely exit Go, the presence of cyclin G1 would be minimal. Furthermore, since the Go phase is associated with cells that have exited the cell cycle, key regulators of the cell cycle, such as cyclin G1, do not progress in their usual cycle-dependent manner, leading to a lack of need for accumulation.
It is essential to note that the accumulation of cyclins is typically tightly regulated and linked to cell cycle progression, which isn't ongoing in cells stuck in Go. Thus, without the 'go-ahead' signals often provided by external factors like growth hormones or the need for tissue repair due to the death of neighboring cells, there would be little to no stimulus for cyclin G1 accumulation. As a result, in infrequently dividing cells, cyclin G1 levels would be expected to remain low unless specific signals prompt these cells to re-enter the cell cycle.