Question

How do OCPs decrease s/s of primary dysmenorrhea?

a) By reducing uterine blood flow
b) By preventing ovulation
c) By increasing prostaglandin production
d) By inducing menopause

Answer 1

OCPs decrease the symptoms of primary dysmenorrhea by preventing ovulation, which leads to a reduction in prostaglandin production and therefore reduces the intensity of uterine contractions associated with pain.

Step-by-step explanation:

OCPs (Oral Contraceptive Pills) are known to decrease the symptoms of primary dysmenorrhea primarily by preventing ovulation. This action helps manage dysmenorrhea because it results in reduced production of prostaglandins, which are the substances that cause the uterus to contract and can be associated with pain. OCPs typically contain both estrogen and progesterone, which work to stabilize the endometrial lining and reduce the intensity of uterine contractions.

Furthermore, OCPs maintain elevated levels of these hormones, which feedback to the hypothalamus and pituitary to suppress the secretion of FSH (Follicle Stimulating Hormone) and LH (Luteinizing Hormone), thus preventing ovulation. By doing so, they also decrease the levels of estrogens and progesterone that fluctuate during the menstrual cycle, leading to a decrease in the production of prostaglandins and a reduced intensity of menstrual cramps.