Final answer:
Aminoglycosides are known to cause ototoxicity rather than disruption of CN I, which is the olfactory nerve. They function by binding to the 30S subunit of bacterial ribosomes, resulting in the production of faulty proteins that disrupt the bacterial cytoplasmic membrane.
Step-by-step explanation:
The question pertains to the ototoxic effects of an aminoglycoside antibiotic and its disruption of cranial nerve I (CN I), which is the olfactory nerve responsible for the sense of smell. Although aminoglycosides, as a class, are more commonly associated with damage to the ear (ototoxicity) and kidney (nephrotoxicity) as well as the nervous system (neurotoxicity), the mention of CN I disruption is not typically associated with this class of antibiotics. Instead, these drugs are known to bind to the 30S subunit of bacterial ribosomes, leading to the production of faulty proteins that insert into and disrupt the cytoplasmic membrane, thus killing bacterial cells. However, no specific aminoglycoside is recognized for targeting CN I, so it might be a misunderstanding or miscommunication.